CVD
is called ‘colour blindness’ by mistake. In actual fact
the term describes a number of problems that people may have with
their colour vision. Abnormal colour vision may vary from not being
able to distinguish between certain shades of colour to not being
able to identify any colour.
Vision
is truly an amazing process. Receptors in the Retina at the back
of the eye send signals to the brain via the Optic Nerve to be interpreted
as sight. There are two types of receptors - Rods and Cones. Rods
work best in low light levels, found in the outer regions of the
Retina and help with our night and side vision. They are not colour
sensitive. There are three types of Cones responsible for the colour
signals, each containing a different pigment, and they are activated
when the pigments in them absorb light. They are concentrated mainly
in the small area called the Macula, which also gives us our central,
detailed vision. Each has a Latin name relating to the order of
wavelength to which they respond.
Protos:
most receptive to the longer wavelengths (L cones) Reddish colours.
Deuterous:
most receptive to medium wavelengths (M cones) Greenish colours.
Tritos:
most receptive to shorter wavelengths (S cones) Bluish colours.
The
different kinds of CVD result from one or more of the different
cone systems either not functioning at all, or functioning in an
unusual way. The most common forms of CVD result from problems with
either the middle or long wavelength sensitive cone systems and
involve difficulties in discriminating the reds and greens and all
the various shades of these colours.
Protanomaly:
Mild deficiency in the sensitivity of the L cones. Being less sensitive
to red light which will cause a darkening of reds to appear brown
or even black in some cases.
Protanopia::
Total lack of the L cones. Distinguishing shades of colours in the
green-yellow-red section of the spectrum is greatly reduced.
Deuteranomaly:
A mild deficiency in the sensitivity of M cones. The medium wavelength
pigment is shifted towards the red end of the spectrum resulting
in a reduction in sensitivity to the green area of the spectrum.
Tritanomaly:
Mild deficiency is the sensitivity of the S cones. Blues and yellows
will be confused.
Trianopia:
Total lack of the S cones causing confusion in the blue-yellow section
of the spectrum.
Who
does CVD affect?
A general consensus is 8% of the male population and 0.5% of females
have some form of CVD. This generally runs in families, being an
inherited condition. Less commonly, it can be an acquired defect
through conditions such as Diabetes, Cataracts or Optic Neuritis
or can be drug induced.
Our
testing procedure involves first determining the type and degree
of deficiency. We do this with various methods including computer
based tests that each person will perform themselves. The time taken
will vary but we always allow at least pne hour, since after the
correct lens is determined the person will wear the lenses to see
how they change and enhance their world of colours. We also encourage
each person to bring along a family member or someone close to them
to observe the test, as this is the defining moment when true colours
are seen for the first time. Many questions can arise and it is
a great time to share.
After
the analysis procedure, if you are happy with the results, the lenses
will be ordered for you. The lenses are available in non prescription
only. For prescription spectacle wearers, a custom fitting clip-on
is best and can be chosen with one of our fully trained Optical
Dispensers.
The
lenses are by far the best quality lenses to treat CVD. They give
a truer perception of the colours and are very durable.
Hereditary
Chart
Colour
Vision Deficency can be either an inherited or an acquired condition.
8%
of the male population and 0.5% of the female population suffer
from inherited CVD. The genetic heritage is linked to a defect in
the X chromosome and usually affects every second generation of
a family. Diabetes, alcoholism or certain medicines may cause acquired
CVD.

Technical
Features
This
chart shows how the lenses change the spectrum of light which stimulates
the cones to begin releasing the chemicals that produce the electrical
signal that travels along the Optic Nerve up to the Occipital Cortex
in the brain, which is then interpreted as vision, specifically
colour vision.

The
lenses were seven years in the research and development stage. They
are manufactured in special vacuum coating chambers.
Several layers are applied in the correct sequence, which is a patented
process. Each layer must be perfect before the the next layer is
applied and so if one layer does not adhere exactly then the whole
process has to begin again with new lenses. Each batch is thoroughly
inspected and must pass strict quality controls before being allowed
out of the factory.
As the process is complex, only piano or non prescription lenses
are produced and so for spectacle wearers the lenses are put into
clip-ons over the prescription spectacles.

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